A Phase 1/2 Study Evaluating Gene Therapy by Transplantation of Autologous CD34+ Stem Cells Modified Ex Vivo Using Prime Editing (PM359) in Participants With Autosomal Recessive Chronic Granulomatous Disease Due to Mutations in the NCF1 Gene

Status: Active_not_recruiting
Location: See all (5) locations...
Intervention Type: Biological
Study Type: Interventional
Study Phase: Phase 1/Phase 2
SUMMARY

This is an open-label, single-arm, multicenter Phase 1/2 study evaluating the safety and efficacy of gene therapy by transplantation of Prime Edited autologous CD34+ stem cells modified ex vivo (PM359) in participants with autosomal recessive Chronic Granulomatous Disease (CGD) caused by mutations in the NCF1 (Neutrophil Cytosolic Factor 1) gene.

Eligibility
Participation Requirements
Sex: All
Minimum Age: 6
Healthy Volunteers: f
View:

• Autosomal recessive Chronic Granulomatous Disease due to the delGT mutation in NCF1 causing dysfunction of p47phox

• Treated and followed for at least the past 2 years in a specialized center

• Willingness to participate in this study as well as a long-term follow-up study with the understanding that the total participation is 15 years

• At least 1 prior severe CGD-related infection OR an ongoing severe CGD-related infection requiring therapy or that is refractory to standard therapy; OR an autoimmune or inflammatory condition related to CGD that is active or requiring therapy to maintain remission.

Locations
United States
California
University of California Los Angeles Medical Center
Los Angeles
Maryland
NIH Clinical Center
Bethesda
Tennessee
The Children's Hospital at Tristar Medical Group/Sarah Cannon Center for Blood Cancers
Nashville
Other Locations
Canada
CHU - Sainte Justine Hospital
Montreal
United Kingdom
University College of London Hospital
London
Time Frame
Start Date: 2024-10-17
Completion Date: 2030-02
Participants
Target number of participants: 12
Treatments
Experimental: PM359
PM359 is an autologous CD34+ hematopoietic stem cell (HSC) suspension that is Prime Edited at the NCF1 locus resulting in expression of the p47phox protein.
Related Therapeutic Areas
Sponsors
Leads: Prime Medicine, Inc.

This content was sourced from clinicaltrials.gov